Fasting Supplements

I get vitamins from the EAS Shake. See my comment on this post --> I'm so hungry

I also chew 1-2 Flintstones Children's Chewable for topical (throat) B complex with C and 1/2 of a 500 mg Costco chewable Vitamin C (bite it in half). Reference: The Fat Switch

I usually take 3 Costco Ca, Mg and Zinc pills with Vitamin D3 (like Citracal) when eating regularly, but reduce that to 2 after a few fasting days.

I take 4 200 mg Advil once or twice a day, depending on inflammation (arthritis).

I take 400 mg Magnesium Oxide despite stories that "it is not absorbed."

I take 1 Complete Multivitamin (like Centrum) for Active Adults 50+ from Walmart.

I take 4 Docusate Sodium, 100mg (Costco) as a stool softener (like Colace).

I take 4 1200 mg max strength Omega 3 (430 mg EPA, 215 mg DHA) on a normal (Omega 6 rich diet) for balance. I reduce this to 2 or 3 per day after the first few days of the fast. Old References: The Omega Rx Zone and Toxic Fat by Barry Sears

I use Trident orange flavored sugar free gum with Xylitol to freshen my mouth and help with keto breath.

I take Allopurinol 300 mg for the hyperuricemia of fasting (requested from my physician.) (Reference: The Fat Switch)

I take 2.5 mg of liquid Melatonin (with ethyl alcohol) to counteract the caffeine from the decaf coffee to help sleep at night.

I take these mostly at night. I have done 3 day, 5 day and one 10 day "only water" fast on this regimen. Don't recall ever having the "keto flu."

> I'm not sure why LPS would be implicated in my scenario, when I am still and always have been extremely lean, don't exhibit any signs of inflammation or permeable gut barrier, nor any signs of leptin resistance.

Are you sure you can rule it out? With MetS, symptoms show up in different orders for different people. It's perfectly normal to show hepatic IR before obesity is ever exhibited. That's how it is for me, showing signs of hepatic IR although also myself also quite lean (8% BF).

> Also, in the study that he references, the diet was not only high in processed saturated fats, but also processed carbs. My diet was 100% unprocessed & low in carbs. In other words, it's like comparing a paleo diet to SAD – both with high amounts of fat – and considering them to be the same thing.

I just wanted you to see that link because I was very excited to see Guyenet coming around to paying attention to resistant starch and fermentable fiber. But, you know, I coulda said the same thing about my diet a year ago: "100% unprocessed & low in carbs". But, sadly, my FBS was creeping up and I was sleeping like shit. I changed to this diet and my FBS is now in the low to mid 80s and I sleep through the night. Doesn't seem to matter how much or how little I exercise. I'm at a loss to explain this without invoking gut microbes!

> My genes: rs429358: TT rs7412: CC

So you are e3/e3 - that's good! I'm surprised. I didn't know that e3s could also experience elev LDLc w excess SFA.

> How do you know that this is hepatotoxic? Low-carb researchers / fans would argue that it's not dangerous in the case of physiological insulin resistance. Now, obviously I question that line of reasoning – hence the motivation for this entire post – but playing devil's advocate, how do you know it's putting negative stress on the liver?

Elev FBS is symptomatic of NAFLD. Of course, I can't prove that's what was going on, but if I google "high fat diet-induced hepatic lipotoxicity", I get tons of relevant hits. I think it's a dangerous game these apologists for the high fat diet are playing and the burden of proof should be on them to show it's safe. Like you, my entire motivation for the diet change was so that I could feel safer and not have to worry.

> In the case of my LDL, some would argue that my ratio lends itself to no cause for concern, but I agree with you; I think it should be lower, and have therefore reduced my saturated fat intake, in exchange for more carbs. Now, I'll have to keep a closer eye on my triglycerides.

Here's my last labs at 25% P, 25% F and 50% C, all carbs from unrefined, unprocessed, high RS / fiber sources - TG is not a problem: TC: 186 LDL: 89 HDL: 87 TG: 49

> With all of this said, I'm curious of your opinion of the theory that my FBG numbers are predominantly caused by inadequate caloric intake, too few carbs for my lifestyle, or a combination of the two. Your theory is interesting, and may be correct – or a contributor – but I'd like to know what you think of the calorie/carb/physiological insulin resistance argument, which my gut (ha!) tells me is what I'm facing.

Back when I was low carbing, I pursued that physiological IR thought for quite a while. I was reading everything I could, trying to prove to myself either that it was harmless or that it was a problem. Although I never really felt the case was proven, the more I read, the more concerned I got, to where I finally decided to go to higher carb.

Here's the thing: it was really, really hard to add carbs. My BG would skyrocket and stay up a long time. I could only drop my BG by going for a hard run. It took immense effort to turn this around. So I don't think for me it was a harmless physiological IR. I wouldn't want to offer an opinion about what's going on with you.

I can tell you this: when I get too few calories, I lose weight, it doesn't elev my FBG. With my current diet, my FBG numbers don't budge. Nothing I do now makes them budge. No more FBG drama! No more blood sugar drama. I eat as much slow carbs as I want and my PP BGs go to 120 max! It's a F-ing miracle as far as I'm concerned.

> Why so much Vit C? I'm concerned about levels that high, as they have been shown to reduce the gains made from weight training.

It's part of turning on and off insulin resistance. I found out about it from Johnson's work (see The Fat Switch)

> I'm going to pass on the colonoscopy at this point

I just mentioned that in case the prep I had to do for it had anything to do with the success of the diet change.